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NAD+ supplementation reduces neuroinflammation and cell senescence in a transgenic mouse model of Alzheimer's disease via cGAS-STING.
Hou, Yujun; Wei, Yong; Lautrup, Sofie; Yang, Beimeng; Wang, Yue; Cordonnier, Stephanie; Mattson, Mark P; Croteau, Deborah L; Bohr, Vilhelm A.
Affiliation
  • Hou Y; Section on DNA Repair, National Institute on Aging, NIH, Baltimore, MD 21224.
  • Wei Y; Institute for Regenerative Medicine, Shanghai East Hospital, Shanghai Key Laboratory of Signaling and Disease Research, School of Life Sciences and Technology, Tongji University, Shanghai 200092, People's Republic of China.
  • Lautrup S; Section on DNA Repair, National Institute on Aging, NIH, Baltimore, MD 21224.
  • Yang B; Hubei Key Laboratory of Cell Homeostasis, College of Life Sciences, Wuhan University, Wuhan 430072, People's Republic of China.
  • Wang Y; Section on DNA Repair, National Institute on Aging, NIH, Baltimore, MD 21224.
  • Cordonnier S; Department of Clinical Molecular Biology, University of Oslo and Akershus University Hospital, 1478 Lørenskog, Norway.
  • Mattson MP; Section on DNA Repair, National Institute on Aging, NIH, Baltimore, MD 21224.
  • Croteau DL; Section on DNA Repair, National Institute on Aging, NIH, Baltimore, MD 21224.
  • Bohr VA; Section on DNA Repair, National Institute on Aging, NIH, Baltimore, MD 21224.
Proc Natl Acad Sci U S A ; 118(37)2021 09 14.
Article in En | MEDLINE | ID: mdl-34497121
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Full text: 1 Collection: 01-internacional Database: MEDLINE Main subject: Cellular Senescence / Dietary Supplements / Alzheimer Disease / Neuroinflammatory Diseases / Membrane Proteins / NAD / Nucleotidyltransferases Type of study: Etiology_studies / Prognostic_studies Limits: Animals / Humans Language: En Journal: Proc Natl Acad Sci U S A Year: 2021 Document type: Article
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Full text: 1 Collection: 01-internacional Database: MEDLINE Main subject: Cellular Senescence / Dietary Supplements / Alzheimer Disease / Neuroinflammatory Diseases / Membrane Proteins / NAD / Nucleotidyltransferases Type of study: Etiology_studies / Prognostic_studies Limits: Animals / Humans Language: En Journal: Proc Natl Acad Sci U S A Year: 2021 Document type: Article