Abnormal Flow Conditions Promote Endocardial Fibroelastosis Via Endothelial-to-Mesenchymal Transition, Which Is Responsive to Losartan Treatment.
JACC Basic Transl Sci
; 6(12): 984-999, 2021 Dec.
Article
in En
| MEDLINE
| ID: mdl-35024504
AR, aortic regurgitation; EFE, endocardial fibroelastosis; EndoMT, endothelial-to-mesenchymal transition; GO, gene ontology; HLHS, hypoplastic left heart syndrome; HUEEC, human endocardial endothelial cells; HUVEC, human umbilical vein endothelial cells; LSS, laminar shear stress; LV, left ventricle; congenital heart disease; endocardial endothelial cells; endocardial fibroelastosis; endothelial-to-mesenchymal transition; wall shear stress; α-SMA, alpha-smooth muscle actin
Full text:
1
Collection:
01-internacional
Database:
MEDLINE
Type of study:
Prognostic_studies
Language:
En
Journal:
JACC Basic Transl Sci
Year:
2021
Document type:
Article
Affiliation country:
Estados Unidos
Country of publication:
Estados Unidos