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Compound heterozygous mutations in a mouse model of Leber congenital amaurosis reveal the role of CCT2 in photoreceptor maintenance.
Suga, Akiko; Minegishi, Yuriko; Yamamoto, Megumi; Ueda, Koji; Iwata, Takeshi.
Affiliation
  • Suga A; Division of Molecular and Cellular Biology, National Institute of Sensory Organs, NHO Tokyo Medical Center, Tokyo, Japan.
  • Minegishi Y; Cancer Proteomics Group, Cancer Precision Medicine Center, Japanese Foundation for Cancer Research, Tokyo, Japan.
  • Yamamoto M; Division of Molecular and Cellular Biology, National Institute of Sensory Organs, NHO Tokyo Medical Center, Tokyo, Japan.
  • Ueda K; Cancer Proteomics Group, Cancer Precision Medicine Center, Japanese Foundation for Cancer Research, Tokyo, Japan.
  • Iwata T; Division of Molecular and Cellular Biology, National Institute of Sensory Organs, NHO Tokyo Medical Center, Tokyo, Japan. takeshi.iwata@kankakuki.jp.
Commun Biol ; 7(1): 676, 2024 Jun 03.
Article in En | MEDLINE | ID: mdl-38830954
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Full text: 1 Collection: 01-internacional Database: MEDLINE Main subject: Disease Models, Animal / Leber Congenital Amaurosis / Chaperonin Containing TCP-1 Limits: Animals Language: En Journal: Commun Biol Year: 2024 Document type: Article Affiliation country: Japón Country of publication: Reino Unido
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Full text: 1 Collection: 01-internacional Database: MEDLINE Main subject: Disease Models, Animal / Leber Congenital Amaurosis / Chaperonin Containing TCP-1 Limits: Animals Language: En Journal: Commun Biol Year: 2024 Document type: Article Affiliation country: Japón Country of publication: Reino Unido