Your browser doesn't support javascript.
loading
The multiple endocrine neoplasia type 2B point mutation alters long-term regulation and enhances the transforming capacity of the epidermal growth factor receptor.
Pandit, S D; Donis-Keller, H; Iwamoto, T; Tomich, J M; Pike, L J.
Affiliation
  • Pandit SD; Washington University School of Medicine, Department of Surgery, St. Louis, Missouri 63110, USA.
J Biol Chem ; 271(10): 5850-8, 1996 Mar 08.
Article in En | MEDLINE | ID: mdl-8621456
Subject(s)
Search on Google
Add to My VHL
Print
XML
PubMed Links

Collection: 01-internacional Database: MEDLINE Main subject: Cell Transformation, Neoplastic / Proto-Oncogene Proteins / Point Mutation / Receptor Protein-Tyrosine Kinases / Multiple Endocrine Neoplasia Type 2b / Drosophila Proteins / ErbB Receptors Limits: Animals / Humans Language: En Journal: J Biol Chem Year: 1996 Document type: Article Affiliation country: Estados Unidos Country of publication: Estados Unidos
Search on Google
Add to My VHL
Print
XML
PubMed Links

Collection: 01-internacional Database: MEDLINE Main subject: Cell Transformation, Neoplastic / Proto-Oncogene Proteins / Point Mutation / Receptor Protein-Tyrosine Kinases / Multiple Endocrine Neoplasia Type 2b / Drosophila Proteins / ErbB Receptors Limits: Animals / Humans Language: En Journal: J Biol Chem Year: 1996 Document type: Article Affiliation country: Estados Unidos Country of publication: Estados Unidos