c-Jun functions as a calcium-regulated transcriptional activator in the absence of JNK/SAPK1 activation.

Cruzalegui, F H; Hardingham, G E; Bading, H

Cruzalegui, F H; Hardingham, G E; Bading, H.

Affiliation

Cruzalegui FH; Neurobiology Division, Medical Research Council Laboratory of Molecular Biology, Hills Road, Cambridge CB2 2QH, UK.

EMBO J ; 18(5): 1335-44, 1999 Mar 01.

Article in En | MEDLINE | ID: mdl-10064599

Subject(s)

Calcium-Calmodulin-Dependent Protein Kinases/metabolism; Calcium/metabolism; Mitogen-Activated Protein Kinases; Proto-Oncogene Proteins c-jun/metabolism; Transcriptional Activation/genetics; CREB-Binding Protein; Calcium Channels/metabolism; Calcium-Calmodulin-Dependent Protein Kinases/genetics; Cell Line; Cyclic AMP/metabolism; Enzyme Activation/genetics; Gene Expression Regulation/genetics; Genes, Reporter/genetics; Ion Channel Gating; JNK Mitogen-Activated Protein Kinases; Microscopy, Fluorescence; Mutation; Nuclear Proteins/genetics; Phosphorylation; Recombinant Fusion Proteins; Signal Transduction; Trans-Activators/genetics; Transcription, Genetic/genetics

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Full text: 1 Collection: 01-internacional Database: MEDLINE Main subject: Transcriptional Activation / Calcium / Proto-Oncogene Proteins c-jun / Calcium-Calmodulin-Dependent Protein Kinases / Mitogen-Activated Protein Kinases Type of study: Prognostic_studies Language: En Journal: EMBO J Year: 1999 Document type: Article Affiliation country: United kingdom Country of publication: United kingdom

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