IL-4- and IL-4 receptor-deficient BALB/c mice reveal differences in susceptibility to Leishmania major parasite substrains.

Noben-Trauth, N; Paul, W E; Sacks, D L

Noben-Trauth, N; Paul, W E; Sacks, D L.

Affiliation

Noben-Trauth N; Laboratories ofImmunology and Parasitology, National Institutes of Allergy and Infectious Diseases, National Institutes of Health, Bethesda, MD 20892, USA. mnoben@nih.gov

J Immunol ; 162(10): 6132-40, 1999 May 15.

Article in En | MEDLINE | ID: mdl-10229856

Subject(s)

Interleukin-4/immunology; Leishmania major/immunology; Leishmaniasis, Cutaneous/immunology; Receptors, Interleukin-4/immunology; Animals; Disease Susceptibility; Immunoglobulin Isotypes/blood; Interferon-gamma/immunology; Interleukin-13/immunology; Interleukin-4/genetics; Leishmania major/classification; Leishmaniasis, Cutaneous/pathology; Mice; Mice, Inbred BALB C; Mice, Mutant Strains; Receptors, Interleukin-4/genetics; Species Specificity; Th1 Cells/immunology; Th2 Cells/immunology

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Collection: 01-internacional Database: MEDLINE Main subject: Interleukin-4 / Leishmaniasis, Cutaneous / Leishmania major / Receptors, Interleukin-4 Type of study: Prognostic_studies Limits: Animals Language: En Journal: J Immunol Year: 1999 Document type: Article Affiliation country: United States Country of publication: United States

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