Evidence that Par-4 participates in the pathogenesis of HIV encephalitis.

Kruman, I I; Nath, A; Maragos, W F; Chan, S L; Jones, M; Rangnekar, V M; Jakel, R J; Mattson, M P

Kruman, I I; Nath, A; Maragos, W F; Chan, S L; Jones, M; Rangnekar, V M; Jakel, R J; Mattson, M P.

Affiliation

Kruman II; Sanders-Brown Research Center on Aging, Department of Neurology, Lexington, Kentucky, USA.

Am J Pathol ; 155(1): 39-46, 1999 Jul.

Article in En | MEDLINE | ID: mdl-10393834

Subject(s)

Carrier Proteins/physiology; Encephalitis/virology; HIV Infections; Intracellular Signaling Peptides and Proteins; Animals; Apoptosis/physiology; Apoptosis Regulatory Proteins; Carrier Proteins/genetics; Carrier Proteins/metabolism; Cells, Cultured; Chimera/physiology; Encephalitis/metabolism; Gene Products, tat/antagonists & inhibitors; Gene Products, tat/pharmacology; HIV/genetics; HIV Infections/metabolism; Hippocampus/drug effects; Hippocampus/metabolism; Hippocampus/pathology; Humans; Macaca mulatta/metabolism; Neurons/drug effects; Neurons/metabolism; Oligonucleotides, Antisense/pharmacology; Rats; Rats, Sprague-Dawley; Simian Acquired Immunodeficiency Syndrome/metabolism; tat Gene Products, Human Immunodeficiency Virus

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Full text: 1 Collection: 01-internacional Database: MEDLINE Main subject: Carrier Proteins / HIV Infections / Intracellular Signaling Peptides and Proteins / Encephalitis Type of study: Etiology_studies Limits: Animals / Humans Language: En Journal: Am J Pathol Year: 1999 Document type: Article Affiliation country: United States Country of publication: United States

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