Fas- and tumor necrosis factor receptor 1-dependent but not perforin-dependent pathways cause injury in livers infected with an adenovirus construct in mice.

Liu, Z X; Govindarajan, S; Okamoto, S; Dennert, G

Liu, Z X; Govindarajan, S; Okamoto, S; Dennert, G.

Affiliation

Liu ZX; Department of Molecular Microbiology and Immunology, USC/Norris Comprehensive Cancer Center, University of Southern California School of Medicine, Los Angeles, CA 90033-0800, USA.

Hepatology ; 31(3): 665-73, 2000 Mar.

Article in En | MEDLINE | ID: mdl-10706557

Subject(s)

Adenoviridae Infections/metabolism; Liver/metabolism; Membrane Glycoproteins/metabolism; Receptors, Tumor Necrosis Factor/metabolism; fas Receptor/metabolism; Adenoviridae/genetics; Adenoviridae Infections/pathology; Animals; Gene Expression; Immunity, Cellular; Lac Operon; Liver/pathology; Liver/virology; Membrane Glycoproteins/deficiency; Mice; Mice, Inbred C57BL; Perforin; Pore Forming Cytotoxic Proteins; Time Factors; beta-Galactosidase/biosynthesis

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Collection: 01-internacional Database: MEDLINE Main subject: Membrane Glycoproteins / Receptors, Tumor Necrosis Factor / Adenoviridae Infections / Fas Receptor / Liver Limits: Animals Language: En Journal: Hepatology Year: 2000 Document type: Article Affiliation country: United States

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