Involvement of CD95/Apo1/Fas in cell death after myocardial ischemia.

Jeremias, I; Kupatt, C; Martin-Villalba, A; Habazettl, H; Schenkel, J; Boekstegers, P; Debatin, K M

Jeremias, I; Kupatt, C; Martin-Villalba, A; Habazettl, H; Schenkel, J; Boekstegers, P; Debatin, K M.

Affiliation

Jeremias I; German Cancer Research Center, Heidelberg, Germany.

Circulation ; 102(8): 915-20, 2000 Aug 22.

Article in En | MEDLINE | ID: mdl-10952962

Subject(s)

Apoptosis/physiology; Myocardial Ischemia/pathology; Myocardium/pathology; fas Receptor/physiology; Amino Acid Chloromethyl Ketones/pharmacology; Animals; Apoptosis Regulatory Proteins; Caspase Inhibitors; Caspases/physiology; Cell Death/physiology; Cells, Cultured; Cysteine Proteinase Inhibitors/pharmacology; Fas Ligand Protein; Heart/physiology; In Vitro Techniques; Male; Membrane Glycoproteins/biosynthesis; Membrane Glycoproteins/metabolism; Mice; Mice, Inbred C57BL; Myocardial Ischemia/metabolism; Myocardial Reperfusion Injury/metabolism; Myocardial Reperfusion Injury/pathology; Myocardium/cytology; Myocardium/metabolism; Rats; Rats, Wistar; Solubility; TNF-Related Apoptosis-Inducing Ligand; Tumor Necrosis Factor-alpha/biosynthesis; Tumor Necrosis Factor-alpha/metabolism

Search on Google

Add to My VHL

XML

PubMed Links

Collection: 01-internacional Database: MEDLINE Main subject: Myocardial Ischemia / Apoptosis / Fas Receptor / Myocardium Type of study: Prognostic_studies Limits: Animals Language: En Journal: Circulation Year: 2000 Document type: Article Affiliation country: Germany Country of publication: United States

Search on Google

Add to My VHL

XML

PubMed Links