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Endoglin is overexpressed after arterial injury and is required for transforming growth factor-beta-induced inhibition of smooth muscle cell migration.
Ma, X; Labinaz, M; Goldstein, J; Miller, H; Keon, W J; Letarte, M; O'Brien, E.
Affiliation
  • Ma X; Division of Cardiology, University of Ottawa Heart Institute, Ottawa, Ontario, Canada.
Arterioscler Thromb Vasc Biol ; 20(12): 2546-52, 2000 Dec.
Article in En | MEDLINE | ID: mdl-11116051
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Collection: 01-internacional Database: MEDLINE Main subject: Coronary Artery Disease / Transforming Growth Factor beta / Vascular Cell Adhesion Molecule-1 Type of study: Prognostic_studies Language: En Journal: Arterioscler Thromb Vasc Biol Journal subject: ANGIOLOGIA Year: 2000 Document type: Article Affiliation country: Canada Country of publication: United States
Search on Google
Collection: 01-internacional Database: MEDLINE Main subject: Coronary Artery Disease / Transforming Growth Factor beta / Vascular Cell Adhesion Molecule-1 Type of study: Prognostic_studies Language: En Journal: Arterioscler Thromb Vasc Biol Journal subject: ANGIOLOGIA Year: 2000 Document type: Article Affiliation country: Canada Country of publication: United States