Analysis of the mechanism of action of non-deletion hereditary persistence of fetal hemoglobin mutants in transgenic mice.

Li, Q; Duan, Z J; Stamatoyannopoulos, G

Li, Q; Duan, Z J; Stamatoyannopoulos, G.

Affiliation

Li Q; Division of Medical Genetics, School of Medicine, University of Washington, Seattle, WA 98195, USA. gstam@u.washington.edu

EMBO J ; 20(1-2): 157-64, 2001 Jan 15.

Article in En | MEDLINE | ID: mdl-11226166

Subject(s)

Erythropoiesis/genetics; Fetal Hemoglobin/genetics; Globins/genetics; Hemoglobinopathies/genetics; Promoter Regions, Genetic; Sequence Deletion; Animals; Base Sequence; Embryo, Mammalian; Humans; Mice; Mice, Transgenic; Models, Animal; Phenotype; Point Mutation

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Full text: 1 Collection: 01-internacional Database: MEDLINE Main subject: Fetal Hemoglobin / Globins / Promoter Regions, Genetic / Sequence Deletion / Erythropoiesis / Hemoglobinopathies Limits: Animals / Humans Language: En Journal: EMBO J Year: 2001 Document type: Article Affiliation country: United States

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