DNase I disinhibition is predominantly associated with actin hyperpolymerization after traumatic brain injury.

Bareyre, F M; Raghupathi, R; Saatman, K E; McIntosh, T K

Bareyre, F M; Raghupathi, R; Saatman, K E; McIntosh, T K.

Affiliation

Bareyre FM; Department of Neurosurgery, University of Pennsylvania, Philadelphia, USA.

J Neurochem ; 77(1): 173-81, 2001 Apr.

Article in En | MEDLINE | ID: mdl-11279273

Subject(s)

Actins/metabolism; Brain Injuries/metabolism; Deoxyribonuclease I/metabolism; Animals; Apoptosis; Brain Injuries/pathology; Caspases/metabolism; Deoxyribonuclease I/antagonists & inhibitors; Disease Models, Animal; Hippocampus/metabolism; Hippocampus/pathology; Immunoblotting; Male; Parietal Lobe/metabolism; Parietal Lobe/pathology; Rats; Rats, Sprague-Dawley; Temporal Lobe/metabolism; Temporal Lobe/pathology; Thalamus/metabolism; Thalamus/pathology; Tissue Extracts/pharmacology; Wounds, Nonpenetrating

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Collection: 01-internacional Database: MEDLINE Main subject: Brain Injuries / Actins / Deoxyribonuclease I Type of study: Risk_factors_studies Limits: Animals Language: En Journal: J Neurochem Year: 2001 Document type: Article Affiliation country: United States Country of publication: United kingdom

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