Induction of pRb degradation by the human papillomavirus type 16 E7 protein is essential to efficiently overcome p16INK4a-imposed G1 cell cycle Arrest.

Giarrè, M; Caldeira, S; Malanchi, I; Ciccolini, F; Leão, M J; Tommasino, M

Giarrè, M; Caldeira, S; Malanchi, I; Ciccolini, F; Leão, M J; Tommasino, M.

Affiliation

Giarrè M; Angewandte Tumorvirologie, Deutsches Krebsforschungszentrum, D-69120 Heidelberg, Germany.

J Virol ; 75(10): 4705-12, 2001 May.

Article in En | MEDLINE | ID: mdl-11312342

Subject(s)

Cyclin-Dependent Kinase Inhibitor p16/metabolism; Oncogene Proteins, Viral/metabolism; Papillomaviridae/metabolism; Retinoblastoma Protein/metabolism; 3T3 Cells; Animals; Cell Division; Cyclin-Dependent Kinase Inhibitor p16/genetics; G1 Phase; Humans; Mice; Oncogene Proteins, Viral/genetics; Papillomaviridae/genetics; Papillomavirus E7 Proteins; Recombinant Fusion Proteins/genetics; Recombinant Fusion Proteins/metabolism

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Full text: 1 Collection: 01-internacional Database: MEDLINE Main subject: Papillomaviridae / Oncogene Proteins, Viral / Retinoblastoma Protein / Cyclin-Dependent Kinase Inhibitor p16 Limits: Animals / Humans Language: En Journal: J Virol Year: 2001 Document type: Article Affiliation country: Germany Country of publication: United States

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