Point mutations of K-ras and H-ras genes in forestomach neoplasms from control B6C3F1 mice and following exposure to 1,3-butadiene, isoprene or chloroprene for up to 2-years.

Sills, R C; Hong, H L; Boorman, G A; Devereux, T R; Melnick, R L

Sills, R C; Hong, H L; Boorman, G A; Devereux, T R; Melnick, R L.

Affiliation

Sills RC; Laboratory of Experimental Pathology, Environmental Toxicology Program, National Institute of Environmental Health Sciences, PO Box 12233, Research Triangle Park, NC 27709, USA. sills@niehs.nih.gov

Chem Biol Interact ; 135-136: 373-86, 2001 Jun 01.

Article in En | MEDLINE | ID: mdl-11397402

Subject(s)

Butadienes/toxicity; Chloroprene/toxicity; Genes, ras/drug effects; Hemiterpenes; Pentanes; Point Mutation; Stomach Neoplasms/genetics; Animals; Base Sequence; Butadienes/administration & dosage; Chloroprene/administration & dosage; DNA Damage; DNA Primers/genetics; Female; Humans; Male; Mice; Polymorphism, Restriction Fragment Length; Polymorphism, Single-Stranded Conformational; Stomach Neoplasms/chemically induced; Stomach Neoplasms/pathology; Time Factors

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Collection: 01-internacional Database: MEDLINE Main subject: Pentanes / Stomach Neoplasms / Butadienes / Chloroprene / Genes, ras / Point Mutation / Hemiterpenes Type of study: Prognostic_studies Limits: Animals / Female / Humans / Male Language: En Journal: Chem Biol Interact Year: 2001 Document type: Article Affiliation country: United States Country of publication: Ireland

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