Increased post-traumatic survival of neurons in IL-6-knockout mice on a background of EAE susceptibility.

Fisher, J; Mizrahi, T; Schori, H; Yoles, E; Levkovitch-Verbin, H; Haggiag, S; Revel, M; Schwartz, M

Fisher, J; Mizrahi, T; Schori, H; Yoles, E; Levkovitch-Verbin, H; Haggiag, S; Revel, M; Schwartz, M.

Affiliation

Fisher J; Department of Neurobiology, The Weizmann Institute of Science, 76100, Rehovot, Israel.

J Neuroimmunol ; 119(1): 1-9, 2001 Sep 03.

Article in En | MEDLINE | ID: mdl-11525794

Subject(s)

Interleukin-6/deficiency; Neurons/physiology; Optic Nerve Injuries/physiopathology; Animals; Cell Survival/drug effects; Drug Resistance; Encephalomyelitis, Autoimmune, Experimental/genetics; Female; Genetic Predisposition to Disease; Glutamic Acid/poisoning; Interleukin-6/genetics; Interleukin-6/pharmacology; Macrophages/pathology; Male; Mice; Mice, Inbred C57BL; Mice, Knockout/genetics; Nerve Crush; Nerve Degeneration/etiology; Optic Nerve Injuries/pathology; Retinal Ganglion Cells/pathology; Retinal Ganglion Cells/physiology

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Collection: 01-internacional Database: MEDLINE Main subject: Interleukin-6 / Optic Nerve Injuries / Neurons Type of study: Etiology_studies Limits: Animals Language: En Journal: J Neuroimmunol Year: 2001 Document type: Article Affiliation country: Israel

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