The sea anemone toxins BgII and BgIII prolong the inactivation time course of the tetrodotoxin-sensitive sodium current in rat dorsal root ganglion neurons.

Salceda, Emilio; Garateix, Anoland; Soto, Enrique

Salceda, Emilio; Garateix, Anoland; Soto, Enrique.

Affiliation

Salceda E; Instituto de Fisiología, Universidad Autónoma de Puebla, Pue, México. escalceda@siu.buap.mx

J Pharmacol Exp Ther ; 303(3): 1067-74, 2002 Dec.

Article in En | MEDLINE | ID: mdl-12438529

Subject(s)

Cnidarian Venoms/pharmacology; Ganglia, Spinal/drug effects; Neurons/drug effects; Sodium Channels/physiology; Tetrodotoxin/pharmacology; Animals; Cells, Cultured; Dose-Response Relationship, Drug; Drug Synergism; Female; Ganglia, Spinal/cytology; Ganglia, Spinal/physiology; Male; Neurons/cytology; Neurons/physiology; Rats; Rats, Wistar; Sea Anemones

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Collection: 01-internacional Database: MEDLINE Main subject: Tetrodotoxin / Sodium Channels / Cnidarian Venoms / Ganglia, Spinal / Neurons Type of study: Diagnostic_studies Limits: Animals Language: En Journal: J Pharmacol Exp Ther Year: 2002 Document type: Article Country of publication: United States

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