Histone H2AX: a dosage-dependent suppressor of oncogenic translocations and tumors.

Bassing, Craig H; Suh, Heikyung; Ferguson, David O; Chua, Katrin F; Manis, John; Eckersdorff, Mark; Gleason, Megan; Bronson, Rodrick; Lee, Charles; Alt, Frederick W

Bassing, Craig H; Suh, Heikyung; Ferguson, David O; Chua, Katrin F; Manis, John; Eckersdorff, Mark; Gleason, Megan; Bronson, Rodrick; Lee, Charles; Alt, Frederick W.

Affiliation

Bassing CH; Howard Hughes Medical Institute, The Children's Hospital, Department of Genetics, Harvard Medical School and The Center for Blood Research, Boston, MA 02115, USA.

Cell ; 114(3): 359-70, 2003 Aug 08.

Article in En | MEDLINE | ID: mdl-12914700

Subject(s)

Histones/metabolism; Neoplasms/genetics; Oncogenes; Translocation, Genetic; Tumor Suppressor Protein p53/genetics; Tumor Suppressor Proteins/metabolism; Animals; Base Sequence; Chromatin; DNA Damage; DNA Repair; Gene Targeting; Histones/genetics; Humans; In Situ Hybridization, Fluorescence; Karyotyping; Lymphoma, B-Cell/genetics; Lymphoma, B-Cell/metabolism; Mice; Mice, Inbred C57BL; Mice, Knockout; Neoplasms/metabolism; Sequence Alignment; Survival Rate; Tumor Suppressor Protein p53/metabolism; Tumor Suppressor Proteins/genetics

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Collection: 01-internacional Database: MEDLINE Main subject: Oncogenes / Translocation, Genetic / Histones / Tumor Suppressor Protein p53 / Tumor Suppressor Proteins / Neoplasms Limits: Animals / Humans Language: En Journal: Cell Year: 2003 Document type: Article Affiliation country: United States Country of publication: United States

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