Camptothecin induced mitochondrial dysfunction leading to programmed cell death in unicellular hemoflagellate Leishmania donovani.

Sen, N; Das, B B; Ganguly, A; Mukherjee, T; Tripathi, G; Bandyopadhyay, S; Rakshit, S; Sen, T; Majumder, H K

Sen, N; Das, B B; Ganguly, A; Mukherjee, T; Tripathi, G; Bandyopadhyay, S; Rakshit, S; Sen, T; Majumder, H K.

Affiliation

Sen N; Division of Infectious Diseases, Indian Institute of Chemical Biology. 4, Raja SC Mullick Road, Kolkata 700 032, India.

Cell Death Differ ; 11(8): 924-36, 2004 Aug.

Article in En | MEDLINE | ID: mdl-15118764

Subject(s)

Apoptosis/physiology; Camptothecin/pharmacology; Caspases/metabolism; Leishmania donovani/metabolism; Mitochondria/metabolism; Animals; Apoptosis/drug effects; Caspase 3; Cytochrome c Group/metabolism; DNA Fragmentation; DNA Topoisomerases, Type I/metabolism; Enzyme Inhibitors/pharmacology; Humans; Leishmania donovani/ultrastructure; Mitochondria/ultrastructure; Reactive Oxygen Species/metabolism; Topoisomerase I Inhibitors; Tumor Cells, Cultured

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Collection: 01-internacional Database: MEDLINE Main subject: Leishmania donovani / Camptothecin / Apoptosis / Caspases / Mitochondria Limits: Animals / Humans Language: En Journal: Cell Death Differ Year: 2004 Document type: Article Affiliation country: India Country of publication: United kingdom

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