Subthalamic nucleus lesion reverses motor abnormalities and striatal glutamatergic overactivity in experimental parkinsonism.

Centonze, D; Gubellini, P; Rossi, S; Picconi, B; Pisani, A; Bernardi, G; Calabresi, P; Baunez, C

Centonze, D; Gubellini, P; Rossi, S; Picconi, B; Pisani, A; Bernardi, G; Calabresi, P; Baunez, C.

Affiliation

Centonze D; Clinica Neurologica, Dipartimento di Neuroscienze, Università di Roma Tor Vergata, Via Montpellier 1, 00133 Rome, Italy. centonze@uniroma2.it

Neuroscience ; 133(3): 831-40, 2005.

Article in En | MEDLINE | ID: mdl-15893432

Subject(s)

Corpus Striatum/physiology; Movement Disorders/physiopathology; Parkinson Disease/physiopathology; Subthalamic Nucleus/physiology; Animals; Antiparkinson Agents/pharmacology; Corpus Striatum/pathology; Denervation; Glutamic Acid/physiology; Levodopa/pharmacology; Male; Movement Disorders/drug therapy; Movement Disorders/pathology; Neurons/pathology; Neurons/physiology; Oxidopamine/pharmacology; Parkinson Disease/drug therapy; Parkinson Disease/pathology; Rats; Rats, Wistar; Subthalamic Nucleus/pathology; Sympatholytics/pharmacology; Synaptic Transmission/drug effects; Synaptic Transmission/physiology

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Collection: 01-internacional Database: MEDLINE Main subject: Parkinson Disease / Corpus Striatum / Subthalamic Nucleus / Movement Disorders Limits: Animals Language: En Journal: Neuroscience Year: 2005 Document type: Article Affiliation country: Italy Country of publication: United States

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