Stathmin-deficient mice develop fibrosis and show delayed recovery from ischemic-reperfusion injury.

Zahedi, Kamyar; Revelo, Monica P; Barone, Sharon; Wang, Zhaohui; Tehrani, Kathy; Citron, David P; Bissler, John J; Rabb, Hamid; Soleimani, Manoocher

Zahedi, Kamyar; Revelo, Monica P; Barone, Sharon; Wang, Zhaohui; Tehrani, Kathy; Citron, David P; Bissler, John J; Rabb, Hamid; Soleimani, Manoocher.

Affiliation

Zahedi K; Division of Nephrology and Hypertension, Children's Hospital Medical Center, Cincinnati, OH 45229-3039, USA. Kamyar.Zahedi@cchmc.org

Am J Physiol Renal Physiol ; 290(6): F1559-67, 2006 Jun.

Article in En | MEDLINE | ID: mdl-16434570

Subject(s)

Kidney/blood supply; Reperfusion Injury/physiopathology; Stathmin/deficiency; Stathmin/physiology; Animals; Apoptosis; Cell Division; DNA Repair; Epithelial Cells/chemistry; Epithelial Cells/pathology; Fibrosis; Inbreeding; Kidney/pathology; Kidney/physiopathology; Kidney Tubules/pathology; Mice; Mice, Inbred C57BL; Mice, Knockout; Proliferating Cell Nuclear Antigen/analysis; Renal Insufficiency/epidemiology; Renal Insufficiency/etiology; Reperfusion Injury/mortality; Reperfusion Injury/pathology; Stathmin/genetics; Weight Loss

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Collection: 01-internacional Database: MEDLINE Main subject: Reperfusion Injury / Stathmin / Kidney Type of study: Etiology_studies Limits: Animals Language: En Journal: Am J Physiol Renal Physiol Journal subject: FISIOLOGIA / NEFROLOGIA Year: 2006 Document type: Article Affiliation country: United States

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