Mitochondrial dysfunction induces aberrant insulin signalling and glucose utilisation in murine C2C12 myotube cells.

Lim, J H; Lee, J I; Suh, Y H; Kim, W; Song, J H; Jung, M H

Lim, J H; Lee, J I; Suh, Y H; Kim, W; Song, J H; Jung, M H.

Affiliation

Lim JH; Division of Metabolic Disease, Department of Biomedical Science, National Institute of Health, 5 Nokbun-dong, Eunpyung-gu, Seoul 122-701, South Korea.

Diabetologia ; 49(8): 1924-36, 2006 Aug.

Article in En | MEDLINE | ID: mdl-16736133

Subject(s)

Glucose/metabolism; Insulin/physiology; Mitochondria, Muscle/physiology; Myoblasts/physiology; Animals; Cell Line; Glucose Transporter Type 4/genetics; Glucose Transporter Type 4/metabolism; Glycolysis; Membrane Potentials; Mice; Mitochondrial Membranes/physiology; Muscle, Skeletal/physiology; Signal Transduction; Transfection; Triglycerides/metabolism

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Collection: 01-internacional Database: MEDLINE Main subject: Myoblasts / Glucose / Insulin / Mitochondria, Muscle Limits: Animals Language: En Journal: Diabetologia Year: 2006 Document type: Article Affiliation country: South Korea Country of publication: Germany

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