Endothelial STAT3 is essential for the protective effects of HO-1 in oxidant-induced lung injury.

Zhang, Xuchen; Shan, Peiying; Jiang, Ge; Zhang, Samuel S-M; Otterbein, Leo E; Fu, Xin-Yuan; Lee, Patty J

Zhang, Xuchen; Shan, Peiying; Jiang, Ge; Zhang, Samuel S-M; Otterbein, Leo E; Fu, Xin-Yuan; Lee, Patty J.

Affiliation

Zhang X; Section of Pulmonary and Critical Care Medicine, Yale University School of Medicine, P.O. Box 208057, New Haven, CT 06520-8057, USA.

FASEB J ; 20(12): 2156-8, 2006 Oct.

Article in En | MEDLINE | ID: mdl-16971418

Subject(s)

Endothelial Cells/pathology; Heme Oxygenase-1/physiology; Hyperoxia/pathology; Lung/pathology; Oxygen/adverse effects; STAT3 Transcription Factor/physiology; Animals; Carbon Monoxide; Heme Oxygenase-1/deficiency; Heme Oxygenase-1/genetics; Humans; Lung Diseases/chemically induced; Lung Diseases/pathology; Mice; Mice, Knockout; RNA, Small Interfering/pharmacology; STAT3 Transcription Factor/deficiency; STAT3 Transcription Factor/genetics; Transfection

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Collection: 01-internacional Database: MEDLINE Main subject: Oxygen / Hyperoxia / Endothelial Cells / Heme Oxygenase-1 / STAT3 Transcription Factor / Lung Limits: Animals / Humans Language: En Journal: FASEB J Journal subject: BIOLOGIA / FISIOLOGIA Year: 2006 Document type: Article Affiliation country: United States Country of publication: United States

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