Positive end-expiratory pressure does not compromise myocardial contractility in myocardial ischemia/reperfusion.

Kubitz, Jens Christian; Annecke, Thorsten; Hinkel, Rabea; Reuter, Daniel Arnulf; Kronas, Nils; Forkl, Steffi; Boekstegers, Peter; Goetz, Alwin Eduard; Kemming, Gregor Iwan

Kubitz, Jens Christian; Annecke, Thorsten; Hinkel, Rabea; Reuter, Daniel Arnulf; Kronas, Nils; Forkl, Steffi; Boekstegers, Peter; Goetz, Alwin Eduard; Kemming, Gregor Iwan.

Affiliation

Kubitz JC; Department of Anesthesiology, Ludwig Maximilians University Munich, Klinikum Grosshadern, Munich, Germany. jkubitz@gmx.de

Shock ; 27(6): 638-43, 2007 Jun.

Article in En | MEDLINE | ID: mdl-17505303

Subject(s)

Myocardial Ischemia/pathology; Positive-Pressure Respiration; Reperfusion Injury; Animals; Blood Pressure; Cardiac Output; Heart Ventricles/pathology; Muscle Contraction; Myocardial Contraction; Myocardium/pathology; Pressure; Stroke; Swine; Time Factors; Water/chemistry

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Collection: 01-internacional Database: MEDLINE Main subject: Reperfusion Injury / Positive-Pressure Respiration / Myocardial Ischemia Type of study: Prognostic_studies Limits: Animals Language: En Journal: Shock Journal subject: ANGIOLOGIA / CARDIOLOGIA Year: 2007 Document type: Article Affiliation country: Germany Country of publication: United States

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