DNA damage induces alternative lengthening of telomeres (ALT) associated promyelocytic leukemia bodies that preferentially associate with linear telomeric DNA.

Fasching, Clare L; Neumann, Axel A; Muntoni, Alessandra; Yeager, Thomas R; Reddel, Roger R

Fasching, Clare L; Neumann, Axel A; Muntoni, Alessandra; Yeager, Thomas R; Reddel, Roger R.

Affiliation

Fasching CL; Children's Medical Research Institute, Westmead, and University of Sydney, New South Wales, Sydney, Australia.

Cancer Res ; 67(15): 7072-7, 2007 Aug 01.

Article in En | MEDLINE | ID: mdl-17652140

Subject(s)

Cell Nucleus Structures/metabolism; DNA Damage/drug effects; Intranuclear Inclusion Bodies/metabolism; Telomere/metabolism; Cell Cycle Proteins/metabolism; Cell Nucleus Structures/genetics; Chromosomes, Human; DNA Repair; Electrophoresis, Gel, Two-Dimensional; Humans; Hydrogen Peroxide/pharmacology; Intranuclear Inclusion Bodies/genetics; Intranuclear Inclusion Bodies/ultrastructure; Methylnitronitrosoguanidine/pharmacology

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Collection: 01-internacional Database: MEDLINE Main subject: DNA Damage / Telomere / Cell Nucleus Structures / Intranuclear Inclusion Bodies Type of study: Risk_factors_studies Limits: Humans Language: En Journal: Cancer Res Year: 2007 Document type: Article Affiliation country: Australia Country of publication: United States

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