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Joint effects of HLA, INS, PTPN22 and CTLA4 genes on the risk of type 1 diabetes.
Bjørnvold, M; Undlien, D E; Joner, G; Dahl-Jørgensen, K; Njølstad, P R; Akselsen, H E; Gervin, K; Rønningen, K S; Stene, L C.
Affiliation
  • Bjørnvold M; Institute of Medical Genetics, Faculty Division Ullevål University Hospital, University of Oslo, P.O. Box 1036, Blindern, NO-0315 Oslo, Norway. marit.bjornvold@medisin.uio.no
Diabetologia ; 51(4): 589-96, 2008 Apr.
Article in En | MEDLINE | ID: mdl-18292987
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Full text: 1 Collection: 01-internacional Database: MEDLINE Main subject: Antigens, CD / Diabetes Mellitus, Type 1 / Protein Tyrosine Phosphatase, Non-Receptor Type 22 / HLA Antigens / Insulin Type of study: Diagnostic_studies / Etiology_studies / Prognostic_studies / Risk_factors_studies Limits: Adolescent / Female / Humans / Male Language: En Journal: Diabetologia Year: 2008 Document type: Article Affiliation country: Norway
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Full text: 1 Collection: 01-internacional Database: MEDLINE Main subject: Antigens, CD / Diabetes Mellitus, Type 1 / Protein Tyrosine Phosphatase, Non-Receptor Type 22 / HLA Antigens / Insulin Type of study: Diagnostic_studies / Etiology_studies / Prognostic_studies / Risk_factors_studies Limits: Adolescent / Female / Humans / Male Language: En Journal: Diabetologia Year: 2008 Document type: Article Affiliation country: Norway