The absence of Hck, Fgr, and Lyn tyrosine kinases augments lung innate immune responses to Pneumocystis murina.

Nelson, Michael P; Metz, Allison E; Li, Shaoguang; Lowell, Clifford A; Steele, Chad

Nelson, Michael P; Metz, Allison E; Li, Shaoguang; Lowell, Clifford A; Steele, Chad.

Affiliation

Nelson MP; Department of Medicine, University of Alabama-Birmingham, 1900 University Blvd., THT 437A, Birmingham, AL 35294, USA.

Infect Immun ; 77(5): 1790-7, 2009 May.

Article in En | MEDLINE | ID: mdl-19255189

Subject(s)

Lung/immunology; Pneumocystis/immunology; Proto-Oncogene Proteins c-hck/deficiency; Proto-Oncogene Proteins/deficiency; src-Family Kinases/deficiency; Animals; Cytokines/metabolism; Granulocytes/immunology; Lung/microbiology; Lung/pathology; Macrophages/immunology; Male; Mice; Mice, Inbred C57BL; Microbial Viability

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Full text: 1 Collection: 01-internacional Database: MEDLINE Main subject: Pneumocystis / Proto-Oncogene Proteins / Src-Family Kinases / Proto-Oncogene Proteins c-hck / Lung Limits: Animals Language: En Journal: Infect Immun Year: 2009 Document type: Article Affiliation country: United States Country of publication: United States

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