Estrogen-induced interaction between KLF5 and estrogen receptor (ER) suppresses the function of ER in ER-positive breast cancer cells.

Guo, Peng; Dong, Xue-Yuan; Zhao, Ke-Wen; Sun, Xiaodong; Li, Qunna; Dong, Jin-Tang

Guo, Peng; Dong, Xue-Yuan; Zhao, Ke-Wen; Sun, Xiaodong; Li, Qunna; Dong, Jin-Tang.

Affiliation

Guo P; Department of Hematology and Medical Oncology and Winship Cancer Institute, Emory University School of Medicine, Atlanta, GA 30322, USA.

Int J Cancer ; 126(1): 81-9, 2010 Jan 01.

Article in En | MEDLINE | ID: mdl-19569049

Subject(s)

Breast Neoplasms/metabolism; Estrogen Receptor alpha/physiology; Estrogens/physiology; Kruppel-Like Transcription Factors/physiology; Base Sequence; Breast Neoplasms/pathology; Cathepsin D/genetics; Cell Line, Tumor; Cell Proliferation; Chromatin Immunoprecipitation; DNA Primers; Estrogen Receptor alpha/metabolism; Estrogens/metabolism; Female; Gene Expression Regulation, Neoplastic; Genes, myc; Humans; Kruppel-Like Transcription Factors/metabolism; Polymerase Chain Reaction; Protein Binding; RNA, Small Interfering; Transcription, Genetic/physiology

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Full text: 1 Collection: 01-internacional Database: MEDLINE Main subject: Breast Neoplasms / Estrogen Receptor alpha / Estrogens / Kruppel-Like Transcription Factors Limits: Female / Humans Language: En Journal: Int J Cancer Year: 2010 Document type: Article Affiliation country: United States

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