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CaMKII phosphorylates collapsin response mediator protein 2 and modulates axonal damage during glutamate excitotoxicity.
Hou, Sheng T; Jiang, Susan X; Aylsworth, Amy; Ferguson, Graeme; Slinn, Jacqueline; Hu, Houwen; Leung, Thomas; Kappler, Joachim; Kaibuchi, Kozo.
Affiliation
  • Hou ST; Experimental NeuroTherapeutics Laboratory, Institute for Biological Sciences, National Research Council Canada, Ottawa, Ontario, Canada. sheng.hou@nrc-cnrc.gc.ca
J Neurochem ; 111(3): 870-81, 2009 Nov.
Article in En | MEDLINE | ID: mdl-19735446

Full text: 1 Collection: 01-internacional Database: MEDLINE Main subject: Axons / Glutamic Acid / Intercellular Signaling Peptides and Proteins / Calcium-Calmodulin-Dependent Protein Kinase Type 2 / Nerve Tissue Proteins / Neurons Type of study: Prognostic_studies Limits: Animals Language: En Journal: J Neurochem Year: 2009 Document type: Article Affiliation country: Canada Country of publication: United kingdom

Full text: 1 Collection: 01-internacional Database: MEDLINE Main subject: Axons / Glutamic Acid / Intercellular Signaling Peptides and Proteins / Calcium-Calmodulin-Dependent Protein Kinase Type 2 / Nerve Tissue Proteins / Neurons Type of study: Prognostic_studies Limits: Animals Language: En Journal: J Neurochem Year: 2009 Document type: Article Affiliation country: Canada Country of publication: United kingdom