Dipyridamole increases gap junction coupling in bovine GM-7373 aortic endothelial cells by a cAMP-protein kinase A dependent pathway.

Begandt, D; Bintig, W; Oberheide, K; Schlie, S; Ngezahayo, A

Begandt, D; Bintig, W; Oberheide, K; Schlie, S; Ngezahayo, A.

Affiliation

Begandt D; Institute of Biophysics, Leibniz University Hannover, Herrenhäuserstr. 2, 30419, Hannover, Germany.

J Bioenerg Biomembr ; 42(1): 79-84, 2010 Feb.

Article in En | MEDLINE | ID: mdl-20054624

Subject(s)

Cyclic AMP-Dependent Protein Kinases/metabolism; Dipyridamole/pharmacology; Endothelial Cells/drug effects; Endothelial Cells/metabolism; Gap Junctions/drug effects; Gap Junctions/metabolism; Animals; Cattle; Cell Line; Connexins/metabolism; Cyclic AMP-Dependent Protein Kinases/antagonists & inhibitors; Fibrinolytic Agents/pharmacology; Isoquinolines/pharmacology; Models, Biological; Protein Kinase Inhibitors/pharmacology; Sulfonamides/pharmacology

Fulltext

Add to My VHL

XML

PubMed Links

Search on Google

Full text: 1 Collection: 01-internacional Database: MEDLINE Main subject: Gap Junctions / Cyclic AMP-Dependent Protein Kinases / Endothelial Cells / Dipyridamole Limits: Animals Language: En Journal: J Bioenerg Biomembr Year: 2010 Document type: Article Affiliation country: Germany Country of publication: United States

Fulltext

Add to My VHL

XML

PubMed Links

Search on Google