SR proteins induce alternative exon skipping through their activities on the flanking constitutive exons.

Han, Joonhee; Ding, Jian-Hua; Byeon, Cheol W; Kim, Jee H; Hertel, Klemens J; Jeong, Sunjoo; Fu, Xiang-Dong

Han, Joonhee; Ding, Jian-Hua; Byeon, Cheol W; Kim, Jee H; Hertel, Klemens J; Jeong, Sunjoo; Fu, Xiang-Dong.

Affiliation

Han J; Department of Cellular and Molecular Medicine, University of California, San Diego, La Jolla, CA 92093-0651, USA.

Mol Cell Biol ; 31(4): 793-802, 2011 Feb.

Article in En | MEDLINE | ID: mdl-21135118

Subject(s)

Alternative Splicing; Exons; Nuclear Proteins/metabolism; RNA-Binding Proteins/metabolism; Animals; Base Sequence; Calcium-Calmodulin-Dependent Protein Kinase Type 2/genetics; Calcium-Calmodulin-Dependent Protein Kinase Type 2/metabolism; Cell Differentiation; Cells, Cultured; Mice; Mice, Knockout; Models, Biological; Mutagenesis; Neurons/cytology; Neurons/metabolism; Nuclear Proteins/deficiency; Nuclear Proteins/genetics; RNA-Binding Proteins/genetics; Recombinant Proteins/genetics; Recombinant Proteins/metabolism; Serine-Arginine Splicing Factors

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Full text: 1 Collection: 01-internacional Database: MEDLINE Main subject: Nuclear Proteins / Exons / RNA-Binding Proteins / Alternative Splicing Type of study: Prognostic_studies Limits: Animals Language: En Journal: Mol Cell Biol Year: 2011 Document type: Article Affiliation country: United States Country of publication: United States

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