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ATM damage response and XLF repair factor are functionally redundant in joining DNA breaks.
Zha, Shan; Guo, Chunguang; Boboila, Cristian; Oksenych, Valentyn; Cheng, Hwei-Ling; Zhang, Yu; Wesemann, Duane R; Yuen, Grace; Patel, Harin; Goff, Peter H; Dubois, Richard L; Alt, Frederick W.
Affiliation
  • Zha S; Howard Hughes Medical Institute, The Children's Hospital, the Immune Disease Institute and the Harvard Medical School, Boston, Massachusetts 02115, USA.
Nature ; 469(7329): 250-4, 2011 Jan 13.
Article in En | MEDLINE | ID: mdl-21160472
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Full text: 1 Collection: 01-internacional Database: MEDLINE Main subject: Recombination, Genetic / Histones / Gene Rearrangement, B-Lymphocyte / Protein Serine-Threonine Kinases / Cell Cycle Proteins / Tumor Suppressor Proteins / DNA-Binding Proteins / DNA Repair / DNA Breaks, Double-Stranded Limits: Animals Language: En Journal: Nature Year: 2011 Document type: Article Affiliation country: United States Country of publication: United kingdom
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Full text: 1 Collection: 01-internacional Database: MEDLINE Main subject: Recombination, Genetic / Histones / Gene Rearrangement, B-Lymphocyte / Protein Serine-Threonine Kinases / Cell Cycle Proteins / Tumor Suppressor Proteins / DNA-Binding Proteins / DNA Repair / DNA Breaks, Double-Stranded Limits: Animals Language: En Journal: Nature Year: 2011 Document type: Article Affiliation country: United States Country of publication: United kingdom