D469del-COMP retention in chondrocytes stimulates caspase-independent necroptosis.

Coustry, Françoise; Posey, Karen L; Liu, Peiman; Alcorn, Joseph L; Hecht, Jacqueline T

Coustry, Françoise; Posey, Karen L; Liu, Peiman; Alcorn, Joseph L; Hecht, Jacqueline T.

Affiliation

Coustry F; Department of Pediatrics, University of Texas Medical School at Houston, Houston, TX 77030, USA.

Am J Pathol ; 180(2): 738-48, 2012 Feb.

Article in En | MEDLINE | ID: mdl-22154936

Subject(s)

Apoptosis/physiology; Caspases/physiology; Chondrocytes/metabolism; Extracellular Matrix Proteins/physiology; Glycoproteins/physiology; Oxidative Stress/physiology; Achondroplasia/metabolism; Animals; DNA Damage/physiology; Endoplasmic Reticulum Stress/physiology; Extracellular Matrix Proteins/metabolism; Glycoproteins/metabolism; Matrilin Proteins; Mice; Rats; Reactive Oxygen Species/metabolism; Transfection; Unfolded Protein Response/physiology; Up-Regulation

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Full text: 1 Collection: 01-internacional Database: MEDLINE Main subject: Glycoproteins / Extracellular Matrix Proteins / Apoptosis / Oxidative Stress / Chondrocytes / Caspases Type of study: Prognostic_studies Limits: Animals Language: En Journal: Am J Pathol Year: 2012 Document type: Article Affiliation country: United States

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