Overexpression of apolipoprotein B attenuates pathologic cardiac remodeling and hypertrophy in response to catecholamines and after myocardial infarction in mice.

Råmunddal, Truls; Lindbom, Malin; Täng, Margareta Scharin; Shao, Yangzhen; Borén, Jan; Omerovic, Elmir

Råmunddal, Truls; Lindbom, Malin; Täng, Margareta Scharin; Shao, Yangzhen; Borén, Jan; Omerovic, Elmir.

Affiliation

Råmunddal T; Department of Molecular and Clinical Medicine, Sahlgrenska Academy, University of Gothenburg, Gothenburg, Sweden. truls@wlab.gu.se

Scand J Clin Lab Invest ; 72(3): 230-6, 2012 May.

Article in En | MEDLINE | ID: mdl-22283829

Subject(s)

Apolipoproteins B/biosynthesis; Heart/physiopathology; Myocardial Infarction/physiopathology; Myocardium/metabolism; Animals; Cardiomegaly/chemically induced; Cardiomegaly/metabolism; Cardiomegaly/pathology; Echocardiography; Humans; Isoproterenol; Male; Mice; Mice, Transgenic; Myocardial Infarction/pathology; Ventricular Function, Left/drug effects; Ventricular Remodeling/physiology

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Full text: 1 Collection: 01-internacional Database: MEDLINE Main subject: Apolipoproteins B / Heart / Myocardial Infarction / Myocardium Limits: Animals / Humans / Male Language: En Journal: Scand J Clin Lab Invest Year: 2012 Document type: Article Affiliation country: Sweden Country of publication: United kingdom

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