Your browser doesn't support javascript.
loading
Chromatin modifications induced by the AML1-ETO fusion protein reversibly silence its genomic targets through AML1 and Sp1 binding motifs.
Maiques-Diaz, A; Chou, F S; Wunderlich, M; Gómez-López, G; Jacinto, F V; Rodriguez-Perales, S; Larrayoz, M J; Calasanz, M J; Mulloy, J C; Cigudosa, J C; Alvarez, S.
Affiliation
  • Maiques-Diaz A; Molecular Cytogenetics Group, Human Cancer Genetics programme, Centro Nacional Investigaciones Oncologicas, Centro de Investigaciones de Enfermedades Raras, Madrid, Spain.
Leukemia ; 26(6): 1329-37, 2012 Jun.
Article in En | MEDLINE | ID: mdl-22289984
Subject(s)
Fulltext
Add to My VHL
Print
XML
PubMed Links
Search on Google

Full text: 1 Collection: 01-internacional Database: MEDLINE Main subject: Hematopoietic Stem Cells / Chromatin / Leukemia, Myeloid, Acute / Oncogene Proteins, Fusion / Sp1 Transcription Factor / Core Binding Factor Alpha 2 Subunit Type of study: Prognostic_studies Limits: Humans Language: En Journal: Leukemia Journal subject: HEMATOLOGIA / NEOPLASIAS Year: 2012 Document type: Article Affiliation country: Spain Country of publication: United kingdom
Fulltext
Add to My VHL
Print
XML
PubMed Links
Search on Google

Full text: 1 Collection: 01-internacional Database: MEDLINE Main subject: Hematopoietic Stem Cells / Chromatin / Leukemia, Myeloid, Acute / Oncogene Proteins, Fusion / Sp1 Transcription Factor / Core Binding Factor Alpha 2 Subunit Type of study: Prognostic_studies Limits: Humans Language: En Journal: Leukemia Journal subject: HEMATOLOGIA / NEOPLASIAS Year: 2012 Document type: Article Affiliation country: Spain Country of publication: United kingdom