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DCK is frequently inactivated in acquired gemcitabine-resistant human cancer cells.
Saiki, Yuriko; Yoshino, Yuki; Fujimura, Hiroko; Manabe, Tatsuya; Kudo, Yuki; Shimada, Miki; Mano, Nariyasu; Nakano, Tomohiro; Lee, Yoonha; Shimizu, Shinjiro; Oba, Shinya; Fujiwara, Sho; Shimizu, Hideyuki; Chen, Na; Nezhad, Zhaleh Kashkouli; Jin, Guo; Fukushige, Shinichi; Sunamura, Makoto; Ishida, Masaharu; Motoi, Fuyuhiko; Egawa, Shinichi; Unno, Michiaki; Horii, Akira.
Affiliation
  • Saiki Y; Department of Molecular Pathology, Tohoku University Graduate School of Medicine, Sendai, Japan.
Biochem Biophys Res Commun ; 421(1): 98-104, 2012 Apr 27.
Article in En | MEDLINE | ID: mdl-22490663
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Full text: 1 Collection: 01-internacional Database: MEDLINE Main subject: Pancreatic Neoplasms / Drug Resistance, Neoplasm / Deoxycytidine / Deoxycytidine Kinase / Antimetabolites, Antineoplastic Limits: Humans Language: En Journal: Biochem Biophys Res Commun Year: 2012 Document type: Article Affiliation country: Japan Country of publication: United States
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Full text: 1 Collection: 01-internacional Database: MEDLINE Main subject: Pancreatic Neoplasms / Drug Resistance, Neoplasm / Deoxycytidine / Deoxycytidine Kinase / Antimetabolites, Antineoplastic Limits: Humans Language: En Journal: Biochem Biophys Res Commun Year: 2012 Document type: Article Affiliation country: Japan Country of publication: United States