Chronic and intermittent hypoxia differentially regulate left ventricular inflammatory and extracellular matrix responses.

Ramirez, Trevi A; Jourdan-Le Saux, Claude; Joy, Anne; Zhang, Jianhua; Dai, Qiuxia; Mifflin, Steve; Lindsey, Merry L

Ramirez, Trevi A; Jourdan-Le Saux, Claude; Joy, Anne; Zhang, Jianhua; Dai, Qiuxia; Mifflin, Steve; Lindsey, Merry L.

Affiliation

Ramirez TA; Barshop Institute of Longevity and Aging Studies, The University of Texas Health Science Center at San Antonio, San Antonio, TX 78245, USA.

Hypertens Res ; 35(8): 811-8, 2012 Aug.

Article in En | MEDLINE | ID: mdl-22495609

Subject(s)

Extracellular Matrix/pathology; Hypoxia/physiopathology; Inflammation/pathology; Ventricular Dysfunction, Left/metabolism; Ventricular Dysfunction, Left/pathology; Animals; Blotting, Western; Body Weight; Cell Adhesion Molecules/metabolism; Chronic Disease; Extracellular Matrix/genetics; Gene Expression/genetics; Inflammation/etiology; Inflammation/genetics; Inflammation Mediators/metabolism; Lung/pathology; Male; Myocardium/pathology; Organ Size; Rats; Rats, Sprague-Dawley; Real-Time Polymerase Chain Reaction; Ventricular Dysfunction, Left/etiology; Ventricular Dysfunction, Left/genetics

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Full text: 1 Collection: 01-internacional Database: MEDLINE Main subject: Ventricular Dysfunction, Left / Extracellular Matrix / Inflammation / Hypoxia Type of study: Etiology_studies Limits: Animals Language: En Journal: Hypertens Res Journal subject: ANGIOLOGIA Year: 2012 Document type: Article Affiliation country: United States Country of publication: United kingdom

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