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HIV-1 reverse transcriptase (RT) polymorphism 172K suppresses the effect of clinically relevant drug resistance mutations to both nucleoside and non-nucleoside RT inhibitors.
Hachiya, Atsuko; Marchand, Bruno; Kirby, Karen A; Michailidis, Eleftherios; Tu, Xiongying; Palczewski, Krzysztof; Ong, Yee Tsuey; Li, Zhe; Griffin, Daniel T; Schuckmann, Matthew M; Tanuma, Junko; Oka, Shinichi; Singh, Kamalendra; Kodama, Eiichi N; Sarafianos, Stefan G.
Affiliation
  • Hachiya A; Christopher S. Bond Life Sciences Center, Department of Molecular Microbiology and Immunology, University of Missouri School of Medicine, Columbia, MO 65211, USA.
J Biol Chem ; 287(35): 29988-99, 2012 Aug 24.
Article in En | MEDLINE | ID: mdl-22761416
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Full text: 1 Collection: 01-internacional Database: MEDLINE Main subject: Polymorphism, Genetic / Zidovudine / Reverse Transcriptase Inhibitors / Anti-HIV Agents / Mutation, Missense / Drug Resistance, Viral / HIV Reverse Transcriptase Limits: Animals / Humans Language: En Journal: J Biol Chem Year: 2012 Document type: Article Affiliation country: United States Country of publication: United States
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Full text: 1 Collection: 01-internacional Database: MEDLINE Main subject: Polymorphism, Genetic / Zidovudine / Reverse Transcriptase Inhibitors / Anti-HIV Agents / Mutation, Missense / Drug Resistance, Viral / HIV Reverse Transcriptase Limits: Animals / Humans Language: En Journal: J Biol Chem Year: 2012 Document type: Article Affiliation country: United States Country of publication: United States