Mutant amyloid precursor protein differentially alters adipose biology under obesogenic and non-obesogenic conditions.

Freeman, Linnea R; Zhang, Le; Dasuri, Kalavathi; Fernandez-Kim, Sun-Ok; Bruce-Keller, Annadora J; Keller, Jeffrey N

Freeman, Linnea R; Zhang, Le; Dasuri, Kalavathi; Fernandez-Kim, Sun-Ok; Bruce-Keller, Annadora J; Keller, Jeffrey N.

Affiliation

Freeman LR; Pennington Biomedical Research Center/LSU System, Baton Rouge, Louisiana, United States of America.

PLoS One ; 7(8): e43193, 2012.

Article in En | MEDLINE | ID: mdl-22912823

Subject(s)

Adipose Tissue/metabolism; Amyloid beta-Protein Precursor/genetics; Amyloid beta-Protein Precursor/metabolism; Homeostasis/physiology; Mutation, Missense/genetics; Obesity/metabolism; Adipocytes/physiology; Adipokines/metabolism; Adiposity/physiology; Analysis of Variance; Animals; Blotting, Western; Cloning, Molecular; DNA Primers/genetics; Diet, High-Fat; Enzyme-Linked Immunosorbent Assay; Homeostasis/genetics; Immunohistochemistry; Leptin/metabolism; Mice; Models, Biological; Polymerase Chain Reaction; Reverse Transcriptase Polymerase Chain Reaction

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Full text: 1 Collection: 01-internacional Database: MEDLINE Main subject: Adipose Tissue / Amyloid beta-Protein Precursor / Mutation, Missense / Homeostasis / Obesity Limits: Animals Language: En Journal: PLoS One Journal subject: CIENCIA / MEDICINA Year: 2012 Document type: Article Affiliation country: United States

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