Expression of a dominant negative mutant innexin in identified neurons and glial cells reveals selective interactions among gap junctional proteins.

Yazdani, Neema; Firme, Constantine P; Macagno, Eduardo R; Baker, Michael W

Yazdani, Neema; Firme, Constantine P; Macagno, Eduardo R; Baker, Michael W.

Affiliation

Yazdani N; Section of Cell and Developmental Biology, University of California, San Diego La Jolla, California, 92093, USA.

Dev Neurobiol ; 73(8): 571-86, 2013 Aug.

Article in En | MEDLINE | ID: mdl-23447124

Subject(s)

Connexins/metabolism; Drosophila Proteins/metabolism; Drosophila melanogaster/metabolism; Neuroglia/metabolism; Neurons/metabolism; Amino Acid Sequence; Animals; Cells, Cultured; Connexins/genetics; Drosophila Proteins/genetics; Drosophila melanogaster/genetics; Gap Junctions/genetics; Gap Junctions/metabolism; Leeches/genetics; Leeches/metabolism; Membrane Proteins/genetics; Membrane Proteins/metabolism; Mutant Proteins/genetics; Mutant Proteins/metabolism

Key words

connexin; gap junction; hemichannel; innexon; neuronal circuit; plaque; synaptogenesis

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Full text: 1 Collection: 01-internacional Database: MEDLINE Main subject: Neuroglia / Connexins / Drosophila Proteins / Drosophila melanogaster / Neurons Limits: Animals Language: En Journal: Dev Neurobiol Journal subject: BIOLOGIA / NEUROLOGIA Year: 2013 Document type: Article Affiliation country: United States

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