Possible involvement of A2A and A3 receptors in modulation of insulin secretion and ß-cell survival in mouse pancreatic islets.

Ohtani, M; Oka, T; Ohura, K

Ohtani, M; Oka, T; Ohura, K.

Affiliation

Ohtani M; Department of Pharmacology, Osaka Dental University, 8-1 Kuzuhahanazono-cho, Hirakata, Osaka 573-1121, Japan. otani-m@cc.osaka-dent.ac.jp

Gen Comp Endocrinol ; 187: 86-94, 2013 Jun 15.

Article in En | MEDLINE | ID: mdl-23453966

Subject(s)

Insulin-Secreting Cells/cytology; Insulin-Secreting Cells/metabolism; Insulin/metabolism; Islets of Langerhans/cytology; Islets of Langerhans/metabolism; Receptors, Purinergic P1/metabolism; Adenosine/pharmacology; Animals; Blotting, Western; Cell Line; Cell Proliferation/drug effects; Cell Survival/drug effects; Cricetinae; Glucose/pharmacology; In Situ Nick-End Labeling; Insulin-Secreting Cells/drug effects; Islets of Langerhans/drug effects; Male; Mice; Mice, Inbred C57BL; Purinergic P1 Receptor Antagonists/pharmacology; Rats; Receptors, Purinergic P1/genetics

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Full text: 1 Collection: 01-internacional Database: MEDLINE Main subject: Islets of Langerhans / Receptors, Purinergic P1 / Insulin-Secreting Cells / Insulin Limits: Animals Language: En Journal: Gen Comp Endocrinol Year: 2013 Document type: Article Affiliation country: Japan Country of publication: United States

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