Rac1 mediates load-driven attenuation of mRNA expression of nerve growth factor beta in cartilage and chondrocytes.

Shim, J W; Hamamura, K; Chen, A; Wan, Q; Na, S; Yokota, H

Shim, J W; Hamamura, K; Chen, A; Wan, Q; Na, S; Yokota, H.

Affiliation

Shim JW; Department of Biomedical Engineering, Indiana University-Purdue University Indianapolis, 723 West Michigan Street, Indianapolis, IN 46202, USA. shimjoon@iupui.edu

J Musculoskelet Neuronal Interact ; 13(3): 372-9, 2013 Sep.

Article in En | MEDLINE | ID: mdl-23989259

Subject(s)

Cartilage/metabolism; Chondrocytes/metabolism; Nerve Growth Factor/biosynthesis; Signal Transduction/physiology; Weight-Bearing/physiology; rac1 GTP-Binding Protein/metabolism; Animals; Blotting, Western; Cells, Cultured; Female; Fluorescence Resonance Energy Transfer; Gene Expression Regulation; Humans; Mice; Mice, Inbred C57BL; RNA, Messenger/metabolism; Real-Time Polymerase Chain Reaction; Stress, Mechanical; p38 Mitogen-Activated Protein Kinases/metabolism

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Full text: 1 Collection: 01-internacional Database: MEDLINE Main subject: Signal Transduction / Cartilage / Weight-Bearing / Chondrocytes / Rac1 GTP-Binding Protein / Nerve Growth Factor Limits: Animals / Female / Humans Language: En Journal: J Musculoskelet Neuronal Interact Journal subject: FISIOLOGIA / NEUROLOGIA / ORTOPEDIA Year: 2013 Document type: Article Affiliation country: United States Country of publication: Greece

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