Leptin acts independently of food intake to modulate gut microbial composition in male mice.

Rajala, Michael W; Patterson, Christa M; Opp, Judith S; Foltin, Susan K; Young, Vincent B; Myers, Martin G

Rajala, Michael W; Patterson, Christa M; Opp, Judith S; Foltin, Susan K; Young, Vincent B; Myers, Martin G.

Affiliation

Rajala MW; Department of Internal Medicine, Division of Gastroenterology (M.W.R.), Division of Metabolism, Endocrinology and Diabetes (C.M.P., M.G.M.) and Division of Infectious Disease (J.S.O., S.K.F., V.B.Y.). Department of Microbiology and Immunology (V.B.Y.), Department of Molecular and Integrative Physiology (M.G.M.), and Neuroscience Program (M.G.M.), University of Michigan, Ann Arbor, Michigan.

Endocrinology ; 155(3): 748-57, 2014 Mar.

Article in En | MEDLINE | ID: mdl-24424041

Subject(s)

Gene Expression Regulation; Hyperphagia; Intestinal Mucosa/metabolism; Intestines/microbiology; Leptin/metabolism; Receptors, Leptin/metabolism; Animals; Antimicrobial Cationic Peptides; Eating; Entamoeba histolytica/metabolism; Female; Gene Expression Profiling; Immunohistochemistry; Male; Mice; Mice, Inbred C57BL; Mice, Transgenic; Microbiota; RNA, Messenger/metabolism; Receptors, Leptin/genetics; Signal Transduction

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Full text: 1 Collection: 01-internacional Database: MEDLINE Main subject: Hyperphagia / Gene Expression Regulation / Leptin / Receptors, Leptin / Intestinal Mucosa / Intestines Limits: Animals Language: En Journal: Endocrinology Year: 2014 Document type: Article Country of publication: United States

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