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Soluble, but not transmembrane, TNF-α is required during influenza infection to limit the magnitude of immune responses and the extent of immunopathology.
DeBerge, Matthew P; Ely, Kenneth H; Enelow, Richard I.
Affiliation
  • DeBerge MP; Department of Medicine, Geisel School of Medicine at Dartmouth College, Lebanon, NH 03756; and matthew.deberge@dartmouth.edu.
  • Ely KH; Department of Medicine, Geisel School of Medicine at Dartmouth College, Lebanon, NH 03756; and.
  • Enelow RI; Department of Medicine, Geisel School of Medicine at Dartmouth College, Lebanon, NH 03756; and Department of Microbiology and Immunology, Geisel School of Medicine at Dartmouth College, Lebanon, NH 03756.
J Immunol ; 192(12): 5839-51, 2014 Jun 15.
Article in En | MEDLINE | ID: mdl-24790150
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Full text: 1 Collection: 01-internacional Database: MEDLINE Main subject: Cell Membrane / Tumor Necrosis Factor-alpha / Orthomyxoviridae Infections / CD8-Positive T-Lymphocytes / Receptors, Tumor Necrosis Factor, Type I / Influenza A Virus, H1N1 Subtype Type of study: Prognostic_studies Limits: Animals Language: En Journal: J Immunol Year: 2014 Document type: Article Country of publication: United States
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Full text: 1 Collection: 01-internacional Database: MEDLINE Main subject: Cell Membrane / Tumor Necrosis Factor-alpha / Orthomyxoviridae Infections / CD8-Positive T-Lymphocytes / Receptors, Tumor Necrosis Factor, Type I / Influenza A Virus, H1N1 Subtype Type of study: Prognostic_studies Limits: Animals Language: En Journal: J Immunol Year: 2014 Document type: Article Country of publication: United States