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SV40 utilizes ATM kinase activity to prevent non-homologous end joining of broken viral DNA replication products.
Sowd, Gregory A; Mody, Dviti; Eggold, Joshua; Cortez, David; Friedman, Katherine L; Fanning, Ellen.
Affiliation
  • Sowd GA; Department of Biological Sciences, Vanderbilt University, Nashville, Tennessee, United States of America.
  • Mody D; Department of Biological Sciences, Vanderbilt University, Nashville, Tennessee, United States of America.
  • Eggold J; Department of Biological Sciences, Vanderbilt University, Nashville, Tennessee, United States of America.
  • Cortez D; Department of Biochemistry, Vanderbilt University School of Medicine, Nashville, Tennessee, United States of America.
  • Friedman KL; Department of Biological Sciences, Vanderbilt University, Nashville, Tennessee, United States of America.
  • Fanning E; Department of Biological Sciences, Vanderbilt University, Nashville, Tennessee, United States of America.
PLoS Pathog ; 10(12): e1004536, 2014 Dec.
Article in En | MEDLINE | ID: mdl-25474690
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Full text: 1 Collection: 01-internacional Database: MEDLINE Main subject: Virus Replication / DNA, Viral / Simian virus 40 / DNA Replication / DNA Breaks, Double-Stranded / DNA End-Joining Repair / Ataxia Telangiectasia Mutated Proteins Limits: Humans Language: En Journal: PLoS Pathog Year: 2014 Document type: Article Affiliation country: United States Country of publication: United States
Fulltext
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Print
XML
PubMed Links
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Full text: 1 Collection: 01-internacional Database: MEDLINE Main subject: Virus Replication / DNA, Viral / Simian virus 40 / DNA Replication / DNA Breaks, Double-Stranded / DNA End-Joining Repair / Ataxia Telangiectasia Mutated Proteins Limits: Humans Language: En Journal: PLoS Pathog Year: 2014 Document type: Article Affiliation country: United States Country of publication: United States