The lung is protected from spontaneous inflammation by autophagy in myeloid cells.

Kanayama, Masashi; He, You-Wen; Shinohara, Mari L

Kanayama, Masashi; He, You-Wen; Shinohara, Mari L.

Affiliation

Kanayama M; Department of Immunology, Duke University Medical Center, Durham, NC 27710; and.
He YW; Department of Immunology, Duke University Medical Center, Durham, NC 27710; and.
Shinohara ML; Department of Immunology, Duke University Medical Center, Durham, NC 27710; and Department of Molecular Genetics and Microbiology, Duke University Medical Center, Durham, NC 27710 mari.shinohara@duke.edu.

J Immunol ; 194(11): 5465-71, 2015 Jun 01.

Article in En | MEDLINE | ID: mdl-25911758

Subject(s)

Autophagy/genetics; Lung/immunology; Microtubule-Associated Proteins/genetics; Myeloid Cells/immunology; Pneumonia/genetics; Animals; Anti-Bacterial Agents/administration & dosage; Autophagy/immunology; Autophagy-Related Protein 7; Cells, Cultured; Environmental Exposure/adverse effects; Immunity, Innate/genetics; Immunity, Innate/immunology; Lung/microbiology; Lung/pathology; Macrophages, Alveolar/immunology; Mice; Mice, Inbred C57BL; Mice, Knockout; Mitochondria/immunology; Pneumonia/immunology; Pneumonia/microbiology; Reactive Oxygen Species/antagonists & inhibitors; Reactive Oxygen Species/metabolism; Toll-Like Receptor 4/immunology

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Full text: 1 Collection: 01-internacional Database: MEDLINE Main subject: Pneumonia / Autophagy / Myeloid Cells / Lung / Microtubule-Associated Proteins Limits: Animals Language: En Journal: J Immunol Year: 2015 Document type: Article Country of publication: United States

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