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Cancer cells, on your histone marks, get SETDB1, silence retrotransposons, and go!
Robbez-Masson, Luisa; Tie, Christopher H C; Rowe, Helen M.
Affiliation
  • Robbez-Masson L; Division of Infection and Immunity, University College London, London, England, UK.
  • Tie CHC; Division of Infection and Immunity, University College London, London, England, UK.
  • Rowe HM; Division of Infection and Immunity, University College London, London, England, UK h.rowe@ucl.ac.uk.
J Cell Biol ; 216(11): 3429-3431, 2017 11 06.
Article in En | MEDLINE | ID: mdl-29066607
ABSTRACT
Cancer cells thrive on genetic and epigenetic changes that confer a selective advantage but also need strategies to avoid immune recognition. In this issue, Cuellar et al. (2017. J. Cell Biol https//doi.org/10.1083/jcb.201612160) find that the histone methyltransferase SETDB1 enables acute myeloid leukemia cells to evade sensing of retrotransposons by innate immune receptors.
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Full text: 1 Collection: 01-internacional Database: MEDLINE Main subject: Leukemia, Myeloid, Acute / Interferons Limits: Humans Language: En Journal: J Cell Biol Year: 2017 Document type: Article Affiliation country: United kingdom
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Full text: 1 Collection: 01-internacional Database: MEDLINE Main subject: Leukemia, Myeloid, Acute / Interferons Limits: Humans Language: En Journal: J Cell Biol Year: 2017 Document type: Article Affiliation country: United kingdom