Bax and caspases regulate increased production of mitochondria-derived reactive species in neuronal apoptosis: LACK of A role for depletion of cytochrome c from the mitochondrial electron transport chain.

Kirkland, Rebecca A; Franklin, James L

Kirkland, Rebecca A; Franklin, James L.

Affiliation

Kirkland RA; College of Family and Consumer Sciences, University of Georgia, 1372 Dawson Hall, Athens, GA 30602, USA.
Franklin JL; Department of Pharmaceutical and Biomedical Sciences, The University of Georgia College of Pharmacy, 357 Wilson Pharmacy, Athens, GA 30602, USA.

Biochem Biophys Rep ; 4: 158-168, 2015 Dec.

Article in En | MEDLINE | ID: mdl-29124200

Key words

Apoptosis; BAF, boc-aspartyl(OMe)-fluoromethylketone; Bax; CM-H2 DCFDA, 5-(and-6)-chloromethyl-2',7' dichlorodihydrofluorescein diacetate; Cytochrome c; FCCP, carbonyl cyanide p-trifluoromethoxyphenylhydrazone; IMM, inner mitochondrial membrane; Mitochondria; NGF; NGF, nerve growth factor; O2.-, superoxide; OMM, outer mitochondrial membrane; ROS, reactive oxygen species; RS, reactive species; Reactive Oxygen; TMRM+, tetramethylrhodamine methyl ester; Δψm,, mitochondrial membrane potential

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Full text: 1 Collection: 01-internacional Database: MEDLINE Language: En Journal: Biochem Biophys Rep Year: 2015 Document type: Article Affiliation country: United States Country of publication: Netherlands

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