CASP8 and FADD-Like Apoptosis Regulating Protein/deficiency; Choledocholithiasis/metabolism; Common Bile Duct/surgery; Hepatic Stellate Cells/metabolism; Hepatitis/metabolism; Hepatocytes/metabolism; Liver Cirrhosis/metabolism; Liver/metabolism; Animals; Apoptosis; Bile Acids and Salts/metabolism; Bile Acids and Salts/toxicity; CASP8 and FADD-Like Apoptosis Regulating Protein/genetics; Cells, Cultured; Choledocholithiasis/etiology; Choledocholithiasis/genetics; Choledocholithiasis/pathology; Cytokines/metabolism; Genetic Predisposition to Disease; Hepatic Stellate Cells/drug effects; Hepatic Stellate Cells/pathology; Hepatitis/etiology; Hepatitis/genetics; Hepatitis/pathology; Hepatocytes/drug effects; Hepatocytes/pathology; Inflammation Mediators/metabolism; Ligation; Liver/drug effects; Liver/pathology; Liver Cirrhosis/etiology; Liver Cirrhosis/genetics; Liver Cirrhosis/pathology; Mice, Knockout; Necrosis; Neutrophil Infiltration; Oxidative Stress; Phenotype; Signal Transduction; Time Factors; Transcription Factor RelA/metabolism; Tumor Necrosis Factor alpha-Induced Protein 3/metabolism; p38 Mitogen-Activated Protein Kinases/metabolism

ASK1; JNK; bile duct ligation; cFLIP; cholestasis; hepatocytes; mitochondrial dysfunction; neutrophils; reactive oxygen species