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Cancer mutational burden is shaped by G4 DNA, replication stress and mitochondrial dysfunction.
Bacolla, Albino; Ye, Zu; Ahmed, Zamal; Tainer, John A.
Affiliation
  • Bacolla A; Departments of Cancer Biology and of Molecular and Cellular Oncology, The University of Texas M.D. Anderson Cancer Center, 6767 Bertner Avenue, Houston, TX, 77030, USA. Electronic address: abacolla@mdanderson.org.
  • Ye Z; Departments of Cancer Biology and of Molecular and Cellular Oncology, The University of Texas M.D. Anderson Cancer Center, 6767 Bertner Avenue, Houston, TX, 77030, USA. Electronic address: zye4@mdanderson.org.
  • Ahmed Z; Departments of Cancer Biology and of Molecular and Cellular Oncology, The University of Texas M.D. Anderson Cancer Center, 6767 Bertner Avenue, Houston, TX, 77030, USA. Electronic address: zahmed@mdanderson.org.
  • Tainer JA; Departments of Cancer Biology and of Molecular and Cellular Oncology, The University of Texas M.D. Anderson Cancer Center, 6767 Bertner Avenue, Houston, TX, 77030, USA. Electronic address: jtainer@mdanderson.org.
Prog Biophys Mol Biol ; 147: 47-61, 2019 10.
Article in En | MEDLINE | ID: mdl-30880007
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Full text: 1 Collection: 01-internacional Database: MEDLINE Main subject: DNA Damage / DNA / DNA Replication / G-Quadruplexes / Mutation Type of study: Prognostic_studies Limits: Humans Language: En Journal: Prog Biophys Mol Biol Year: 2019 Document type: Article Country of publication: United kingdom
Fulltext
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Full text: 1 Collection: 01-internacional Database: MEDLINE Main subject: DNA Damage / DNA / DNA Replication / G-Quadruplexes / Mutation Type of study: Prognostic_studies Limits: Humans Language: En Journal: Prog Biophys Mol Biol Year: 2019 Document type: Article Country of publication: United kingdom