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A spontaneous leptin receptor point mutation causes obesity and differentially affects leptin signaling in hypothalamic nuclei resulting in metabolic dysfunctions distinct from db/db mice.
Piattini, Federica; Le Foll, Christelle; Kisielow, Jan; Rosenwald, Esther; Nielsen, Peter; Lutz, Thomas; Schneider, Christoph; Kopf, Manfred.
Affiliation
  • Piattini F; Institute of Molecular Biomedicine, Department Biology, ETH Zürich, Switzerland.
  • Le Foll C; Institute of Veterinary Physiology, Vetsuisse Faculty, University of Zurich, Switzerland.
  • Kisielow J; Institute of Molecular Biomedicine, Department Biology, ETH Zürich, Switzerland.
  • Rosenwald E; Institute of Molecular Biomedicine, Department Biology, ETH Zürich, Switzerland.
  • Nielsen P; Institute of Molecular Biomedicine, Department Biology, ETH Zürich, Switzerland.
  • Lutz T; Institute of Veterinary Physiology, Vetsuisse Faculty, University of Zurich, Switzerland.
  • Schneider C; Institute of Molecular Biomedicine, Department Biology, ETH Zürich, Switzerland.
  • Kopf M; Institute of Molecular Biomedicine, Department Biology, ETH Zürich, Switzerland. Electronic address: Manfred.Kopf@ethz.ch.
Mol Metab ; 25: 131-141, 2019 07.
Article in En | MEDLINE | ID: mdl-31076350
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Full text: 1 Collection: 01-internacional Database: MEDLINE Main subject: Signal Transduction / Point Mutation / Leptin / Receptors, Leptin / Hypothalamus / Obesity Type of study: Etiology_studies Limits: Animals Language: En Journal: Mol Metab Year: 2019 Document type: Article Affiliation country: Switzerland Country of publication: Germany
Fulltext
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Full text: 1 Collection: 01-internacional Database: MEDLINE Main subject: Signal Transduction / Point Mutation / Leptin / Receptors, Leptin / Hypothalamus / Obesity Type of study: Etiology_studies Limits: Animals Language: En Journal: Mol Metab Year: 2019 Document type: Article Affiliation country: Switzerland Country of publication: Germany